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Abstracts from the International Congress of Parkinson’s and Movement Disorders.

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Articles tagged "Mitochondria"

  • 2016 International Congress

    Genome-wide bioinformatic analysis of Parkinson’s disease: A focus on neuronal health maintenance

    R. Qadri, M.A. Faiq, V. Goyal, A.K. Mukhopadhyay (New Delhi, India)

    Objective: To scan the human genome for genes involved in neuronal health maintenance in Parkinson's disease (PD). Background: An enormous number of genes and pathways…
  • 2016 International Congress

    Development of a human neuroblastoma model of pantothenate kinase-associated neurodegeneration

    A. Di Marco, G. Auciello, A. Vecchi, D. Vignone, M.R. Battista, E. Bracacel, F. Bonelli, E. Nizi, E. Monteagudo, M. Beconi (Pomezia, Italy)

    Objective: To establish a stable human neuronal model defective in PanK2. Background: Pantothenate Kinase-Associated Neurodegeneration (PKAN), is caused by mutations in PANK2, which encodes one…
  • 2016 International Congress

    Same cerebellar atrophy pattern in hyper- and hypokinetic movement disorders due to mitochondrial cytopathy

    S.R. Schreglmann, F. Riederer, C. Ganos, G. Kägi, D. Waldvogel, U. Hidding, E. Krasemann, C.R. Baumann, K.P. Bhatia, H.H. Jung, L. Michels (London, United Kingdom)

    Objective: To describe the prevalence and phenotype of movement disorders (MDs) in patients with mitochondrial cytopathy (MC) and corresponding pattern of cerebral atrophy. Background: MCs…
  • 2016 International Congress

    Parkinson’s disease-associated Miro1 mutants cause mitochondrial dysfunction

    D. Grossmann, D. Scheibner, J. Fitzgerald, W. Mätzler, L. Burbulla, R. Krüger (Belvaux, Luxembourg)

    Objective: In the present study we dissected the role of the first PD-associated mutations in RhoT1, the gene encoding for Miro1. Background: Commonly involved pathogenic…
  • 2016 International Congress

    PINK1-dependent clearance of depolarized mitochondria is driven by the UPS and can occur independently of (macro)autophagy

    A. Rakovic, J. Ziegler, C.U. Mårtensson, J. Prasuhn, K. Shurkewitsch, P. König, H.L. Paulson, C. Klein (Luebeck, Germany)

    Objective: To disect the role of macroautophagy in PINK1-/Parkin-dependent removal of depolarized mitochondria. Background: The ubiquitin ligase Parkin and mitochondrial kinase PINK1 function together in…
  • 2016 International Congress

    Defective ubiquitinated mitochondria accumulation in aged Parkinsonian LRRK2R1441G knockin mice

    H.F. Liu, P.W.L. Ho, L.F. Li, G.C.T. Leung, C.S.C. Lam, M.H.W. Kung, D.B. Ramsden, S.L. Ho (Hong Kong, Hong Kong)

    Objective: (1) To determine potential accumulation of defective ubiquitinated mitochondria in the brains of aged LRRK2R1441G knockin (KI) mice and their age-matched wildtype controls; and…
  • 2016 International Congress

    Drp1 inhibition amelioratesα-synuclein-mediated neurodegeneration in rats

    S. Bido, L. Arcuri, M. Helly, R. Fan, K. Tieu, E. Bezard (Bordeaux, France)

    Objective: To provide new insights on the relationships occurring between mitochondria physiology and α-synuclein (α-syn) accumulation in an animal model of progressive neurodegeneration. Background: In…
  • 2016 International Congress

    Mitochondrial-nuclear interplay in single respiratory chain-deficient Parkinson’s disease neurons

    A. Grünewald, K.A. Rygiel, P.D. Hepplewhite, C.M. Morris, M. Picard, D.M. Turnbull (Belvaux, Luxembourg)

    Objective: To explore the role of the mitochondrial DNA (mtDNA) in the development of respiratory chain complex (CI-IV) deficiencies in the substantia nigra (SN) of…
  • 2016 International Congress

    Mitochondrial response precedes intracellular accumulation of α-synuclein in Parkinson’s disease

    I. Milenkovic, E. Dassler, M. Weber, G.G. Kovacs (Vienna, Austria)

    Objective: To investigate the expression of mitochondrial COX-IV during the progression of Parkinson's disease. Background: Parkinson's disease (PD) is a neurodegenerative disease characterized by the…
  • 2016 International Congress

    Mild mitochondrial impairment promotes corticostriatal synaptic plasticity alterations in PINK1 heterozygous knockout mice

    P. Imbriani, G. Martella, G. Madeo, M. Maltese, V. Vanni, E. Ferraro, E.M. Valente, T. Schirinzi, L. Bonanni, J. Shen, N.B. Mercuri, P. Bonsi, A. Pisani (Rome, Italy)

    Objective: To investigate the effects of the exposure to low-dose rotenone of heterozygous PINK1 knockout (PINK1+/-) mice, compared to their wild-type littermates (PINK1+/+), on dopamine-dependent…
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